Why We Feel Pain, and How We Kill it | A Look at Pain & Painkillers

To make us feel uncomfortable.

To help us avoid danger and protect ourselves.

To signal that our body is healing.

To increase our body temperature.

Taste buds

Photoreceptors

Nociceptors

Thermoreceptors

They make the pain receptors less sensitive.

They lower the pain-receiving threshold, making us feel more pain.

They completely block all pain signals.

They cause the pain receptors to heal faster.

It directly heals damaged tissue.

It increases the body's pain threshold.

It blocks pain-signaling COX enzymes.

It produces more arachidonic acid.

Aspirin

Ibuprofen

Acetaminophen

Naproxen Sodium

By reducing inflammation in the body.

By permanently breaking off pain-causing chemicals.

By blocking pain signals to the brain and changing how pain is perceived.

By being spit out by enzymes after a short time.

Heroin

Methadone

Morphine

Oxycodone

Mambalgins are much cheaper to produce.

Mambalgins target pain-sensing nociceptors directly and have fewer side effects.

Mambalgins can cure the underlying cause of pain.

Mambalgins are only effective for very mild pain.